Department of Anesthesiology
Author Title Type [ Year(Asc)]
Filters: Keyword is Kidney  [Clear All Filters]
2010
Zifarelli G, Liantonio A, Gradogna A, Picollo A, Gramegna G, De Bellis M, Murgia AR, Babini E, D Camerino C, Pusch M.  2010.  Identification of sites responsible for the potentiating effect of niflumic acid on ClC-Ka kidney chloride channels.. Br J Pharmacol. 160(7):1652-61.
Gradogna A, Babini E, Picollo A, Pusch M.  2010.  A regulatory calcium-binding site at the subunit interface of CLC-K kidney chloride channels.. J Gen Physiol. 136(3):311-23.
2007
Picollo A, Liantonio A, Babini E, Camerino DConte, Pusch M.  2007.  Mechanism of interaction of niflumic acid with heterologously expressed kidney CLC-K chloride channels.. J Membr Biol. 216(2-3):73-82.
2006
Liantonio A, Picollo A, Babini E, Carbonara G, Fracchiolla G, Loiodice F, Tortorella V, Pusch M, Camerino DConte.  2006.  Activation and inhibition of kidney CLC-K chloride channels by fenamates.. Mol Pharmacol. 69(1):165-73.
2004
Liantonio A, Pusch M, Picollo A, Guida P, De Luca A, Pierno S, Fracchiolla G, Loiodice F, Tortorella P, Camerino DConte.  2004.  Investigations of pharmacologic properties of the renal CLC-K1 chloride channel co-expressed with barttin by the use of 2-(p-Chlorophenoxy)propionic acid derivatives and other structurally unrelated chloride channels blockers.. J Am Soc Nephrol. 15(1):13-20.
2002
Liantonio A, Accardi A, Carbonara G, Fracchiolla G, Loiodice F, Tortorella P, Traverso S, Guida P, Pierno S, De Luca A et al..  2002.  Molecular requisites for drug binding to muscle CLC-1 and renal CLC-K channel revealed by the use of phenoxy-alkyl derivatives of 2-(p-chlorophenoxy)propionic acid.. Mol Pharmacol. 62(2):265-71.
2000
Nimigean CM, Magleby KL.  2000.  Functional coupling of the beta(1) subunit to the large conductance Ca(2+)-activated K(+) channel in the absence of Ca(2+). Increased Ca(2+) sensitivity from a Ca(2+)-independent mechanism.. J Gen Physiol. 115(6):719-36.
1999
Nimigean CM, Magleby KL.  1999.  The beta subunit increases the Ca2+ sensitivity of large conductance Ca2+-activated potassium channels by retaining the gating in the bursting states.. J Gen Physiol. 113(3):425-40.
1996
Kisor DF, Schmith VD, Wargin WA, Lien CA, Ornstein E, Cook DR.  1996.  Importance of the organ-independent elimination of cisatracurium.. Anesth Analg. 83(5):1065-71.
1986
Boyd RM, Nasjletti A, Heerdt PM, Baer PG.  1986.  PGI2 synthesis and excretion in dog kidney: evidence for renal PG compartmentalization.. Am J Physiol. 250(1 Pt 2):F58-65.